The probability of gastric ulcer patients developing gastric cancer is relatively low, but long-term lack of standardized treatment may increase the risk. Gastric ulcer carcinogenesis is mainly related to factors such as Helicobacter pylori infection, recurrent ulcer attacks, and unhealthy lifestyle habits.
Gastric ulcer itself is a benign lesion, and most patients can effectively control the condition through standardized drug treatment and lifestyle adjustments. Helicobacter pylori infection is the most common cause of gastric ulcer and also a carcinogenic factor in gastric cancer. Eradicating this bacterium can significantly reduce the risk of cancer. When taking nonsteroidal anti-inflammatory drugs for a long time or having precancerous lesions such as gastric mucosal atrophy and intestinal metaplasia, it is necessary to strengthen gastroscopy monitoring.
A small number of gastric ulcers may be accompanied by atypical hyperplasia or precancerous states, and the probability of cancer transformation in such patients may increase. In cases with irregular ulcer edges, large diameters, or prolonged non healing, early gastric cancer should be excluded through pathological biopsy. Genetic factors such as a family history of gastric cancer and type A blood type can also affect the progression of cancer, and this population should shorten the interval between gastroscopy examinations. Patients with gastric ulcers should undergo regular gastroscopy follow-up, especially if accompanied by symptoms such as weight loss and vomiting blood, and seek medical attention promptly. Avoid high salt pickled foods in daily life, quit smoking and limit alcohol consumption, and maintain a regular diet. After eradicating Helicobacter pylori treatment, it is recommended to undergo a carbon-13 breath test every 1-2 years. During gastric mucosal repair, it is recommended to supplement vitamins and high-quality protein in moderation.
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