Should I adjust my diet for low levels of gastrin 17?

Low levels of gastrin 17 usually require dietary adjustments. It is recommended to increase protein intake and reduce stimulating foods. Low levels of gastrin 17 may be related to factors such as atrophic gastritis, Helicobacter pylori infection, and long-term use of acid suppressants. A dietary plan should be developed based on specific causes. Gastrin-17 is secreted by G cells in the gastric antrum, and a decrease in its level often reflects reduced gastric acid secretion or gastric mucosal atrophy. The core of dietary adjustment is to reduce the burden on the stomach and promote mucosal repair. High quality proteins such as fish and eggs can provide repair materials, and it is recommended to use steaming and cooking methods. Animal liver and fermented soy products rich in vitamin B12 can help improve gastric mucosal function and can be supplemented in moderation. At the same time, avoid foods that stimulate stomach acid secretion such as chili peppers, alcohol, and strong tea, and reduce the intake of pickled and fried foods. If accompanied by Helicobacter pylori infection, it is necessary to cooperate with a doctor for eradication treatment. Long term use of proton pump inhibitors should be gradually reduced under the guidance of a doctor. You can eat less and more meals in daily life. Choose digestible foods such as millet porridge and yam, and avoid eating acidic fruits on an empty stomach. When cooking, replace animal oil with olive oil and add vegetables rich in carotenoids such as pumpkin and carrot appropriately.

It is recommended to regularly monitor gastrin 17 levels and gastric function indicators, and perform gastroscopy if necessary. Dietary adjustments should last for 3-6 months, in conjunction with regular sleep and emotional management. If there are obvious symptoms such as abdominal distension and anemia, seek medical attention promptly to rule out complications such as pernicious anemia. Pay attention to distinguishing between physiological abnormalities and pathological abnormalities, and avoid self supplementing with gastric acid stimulants.

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