A stroke output of 45ml is within the normal low range. The resting stroke volume of a normal adult is about 60-80ml, and 45ml may be affected by factors such as cardiac function, posture changes, and exercise level, mainly due to weakened cardiac muscle contractility, insufficient blood volume, abnormal autonomic nervous system regulation, long-term lack of exercise, and potential cardiovascular diseases.
1. Weakened myocardial contractility:
A decrease in myocardial contractility directly reduces stroke output. Commonly seen in patients with myocarditis recovery, mild myocardial ischemia, or early heart failure, this group may be accompanied by mild post activity dyspnea. The ejection fraction can be evaluated by echocardiography, and moderate aerobic exercise such as brisk walking and swimming can improve heart function. 2. Insufficient blood volume: Dehydration or blood loss can lead to a decrease in circulating blood volume, resulting in insufficient ventricular filling. Manifesting as thirst and orthostatic hypotension, it is common in people who work at high temperatures, experience diarrhea, or have excessive menstrual flow. It is recommended to drink 1500-2000ml of water daily and supplement with oral rehydration salts if necessary.
3. Abnormal autonomic nervous system regulation:
Excessive vagal tone can inhibit myocardial contractility, which is more common in individuals with long-term anxiety or athletes in a resting state. May be accompanied by a slower heart rate of 50-60 beats per minute, and improving autonomic balance through deep breathing training and regular sleep patterns.
4. Long term lack of exercise:
Sedentary individuals have poor cardiac compensation and lower resting stroke volume. At the beginning of exercise, there may be palpitations and fatigue. It is recommended to start with low-intensity aerobic exercise for 30 minutes three times a week to gradually improve the pumping efficiency of the heart.
5. Potential cardiovascular disease:
Mild heart valve disease or hypertensive heart disease may be characterized by reduced stroke volume in the early stages, usually accompanied by chest tightness and paroxysmal nocturnal dyspnea. Dynamic electrocardiogram and cardiac ultrasound screening are required, and standardized treatment of the primary disease is necessary after diagnosis.
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