The simplest way to reduce high uric acid levels

Uric acid can be reduced by adjusting diet, increasing water intake, controlling weight, moderate exercise, medication intervention, and other methods. Elevated uric acid is mainly related to factors such as abnormal purine metabolism and reduced renal excretion.

1. Adjusting diet:

Reducing the intake of high purine foods is a basic measure to control uric acid. If the purine content in animal organs, thick meat soup, seafood, etc. exceeds 150 milligrams per 100 grams, strict restrictions must be imposed. It is recommended to choose low purine dairy products, eggs, and fresh vegetables, with a daily purine intake controlled below 200 milligrams. Cherries, celery and other foods contain flavonoids that can assist in promoting uric acid excretion.

2. Increase water intake:

Daily water intake should reach 2000-3000 milliliters, which can dilute urine and promote uric acid excretion. It is recommended to drink plain water, light tea or soda water in portions, as alkaline water can help neutralize uric acid. Avoid sugary drinks and alcohol. Beer contains purines and inhibits uric acid excretion, and should be absolutely prohibited.

3. Weight control:

Losing 5% -10% of weight in obese individuals can reduce uric acid levels by 50-100 μ mol/L. Control BMI below 24 through a reasonable diet and exercise, but avoid rapid weight loss that can lead to ketoacidosis. Waist circumference should be less than 90 centimeters for males and less than 85 centimeters for females. Reduced visceral fat can improve metabolic abnormalities.

4. Moderate exercise:

Engage in 150 minutes of low-intensity aerobic exercise per week, such as brisk walking, swimming, cycling, etc. It is advisable to maintain a heart rate of 220% -60% -70% for exercise intensity, in order to avoid intense exercise that leads to lactate accumulation and competitive inhibition of uric acid excretion. Timely replenish water after exercise.

5. Medication intervention:

When uric acid levels remain above 540 μ mol/L or gout attacks occur, medication should be taken under the guidance of a doctor. Allopurinol and febuxostat, which inhibit uric acid production, and benzbromarone, which promotes excretion, are commonly used drugs. Patients with combined renal insufficiency need to adjust the dosage and regularly monitor liver and kidney function during medication. Long term maintenance of uric acid within the ideal range of 300-360 μ mol/L requires comprehensive management. Daily intake of 500mg of vitamin C can promote uric acid excretion, but avoid excessive intake. Regular sleep patterns can help improve metabolic disorders, while staying up late can exacerbate elevated uric acid levels. Chronic diseases such as hypertension and diabetes need to be controlled synchronously, and the interaction of three highs will aggravate the abnormality of uric acid metabolism. If gout symptoms such as joint redness, swelling, heat and pain occur, seek medical attention promptly to avoid joint deformities and kidney function damage caused by uric acid crystal deposition.