After fasting for a few days, there may indeed be a decrease in hunger, which is related to the body's metabolic adaptation mechanism. The weakening of hunger is mainly affected by factors such as stable blood sugar, ketone body energy supply, and reduced gastric capacity, but there are significant individual differences. In the early stages of fasting, the human body will first consume liver glycogen to maintain blood sugar stability, and after about 24-48 hours, it begins to break down fat and produce ketone bodies. Ketones, as alternative energy sources, can inhibit the activity of the appetite center, which is the main reason for the decrease in physiological hunger. Long term fasting can slow down gastric emptying and reduce gastric acid secretion, which can also alleviate hunger signal transmission.
Some individuals may experience abnormal loss of hunger and should be alert to hypoglycemic reactions or metabolic disorders. When diabetes patients, pregnant women, teenagers and other special groups are deprived of food, the body may not be able to start the ketone body energy supply mechanism normally, but may have hypoglycemic symptoms such as dizziness and fatigue. Long term excessive fasting may also lead to a decrease in basal metabolic rate, making it easier to rebound after resuming diet.
During fasting, it is recommended to choose vegetables and fruits rich in dietary fiber to help prolong satiety, such as broccoli, apples, etc. Maintaining moderate low-intensity exercise such as walking daily can maintain metabolic vitality and avoid sudden resumption of high calorie diets. When experiencing symptoms of low blood sugar such as palpitations and hand tremors, fasting should be stopped immediately. Special populations should consult a professional physician before attempting fasting.
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