Low blood crystal osmotic pressure may be caused by excessive water intake, malnutrition, chronic kidney disease, hyponatremia, adrenal cortex dysfunction, etc. It can be improved by limiting water intake, adjusting dietary structure, medication treatment, and other methods.
1. Excessive water content:
Drinking a large amount of water or intravenous fluids in a short period of time can cause blood dilution, a decrease in electrolyte concentration in plasma, and a decrease in crystal osmotic pressure. This condition is more common in cases of excessive hydration or rapid intravenous infusion after exercise, usually manifested as mild dizziness and fatigue, which can be restored by controlling fluid intake and speed.
2. Malnutrition:
Long term insufficient protein and electrolyte intake can affect the balance of plasma colloid osmotic pressure and crystal osmotic pressure. Vegetarians or extreme cannibals may experience hypoalbuminemia accompanied by low sodium and chlorine levels, and need to increase their intake of high-quality proteins such as eggs and fish, and supplement electrolyte preparations if necessary.
3. Chronic kidney disease:
When renal dysfunction occurs, renal tubular reabsorption is impaired, sodium ions are lost in large amounts with urine, and water excretion is obstructed. These patients often have lower limb edema and elevated blood pressure, and should limit their daily water intake to less than 1000 milliliters and use diuretics such as furosemide to regulate water sodium balance. 4. Hyponatremia: Severe diarrhea, vomiting, or improper use of diuretics can cause excessive loss of sodium ions. When the blood sodium concentration is below 135mmol/L, symptoms such as nausea and drowsiness may occur. Mild cases can be corrected by oral saline solution, while severe cases require intravenous supplementation of hypertonic saline solution.
5. Hormonal abnormalities:
Insufficient aldosterone secretion from the adrenal cortex can affect tubular sodium potassium exchange, leading to increased sodium excretion. The typical symptoms of Addison's disease patients are skin pigmentation and hypotension, requiring long-term use of corticosteroid replacement therapy such as hydrocortisone.
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